tabaslab.com

Ira Tabas, MD, PhD, Columbia University

Macrophage apoptosis and defective efferocytosis lead to plaque necrosis, a key feature of clinically dangerous atherosclerotic plaques. In the setting of obesity, insulin resistance, and type 2 diabetes, liver-derived metabolic perturbations and direct effects of insulin resistance on lesional macrophages promote plaque progression. To block the generation of systemic, liver-derived atherosclerotic risk factors in the setting of obesity and type 2 diabetes. Ira Tabas, MD, PhD. 630 West 168th Street.

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Ira Tabas, MD, PhD, Columbia University | tabaslab.com Reviews

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Macrophage apoptosis and defective efferocytosis lead to plaque necrosis, a key feature of clinically dangerous atherosclerotic plaques. In the setting of obesity, insulin resistance, and type 2 diabetes, liver-derived metabolic perturbations and direct effects of insulin resistance on lesional macrophages promote plaque progression. To block the generation of systemic, liver-derived atherosclerotic risk factors in the setting of obesity and type 2 diabetes. Ira Tabas, MD, PhD. 630 West 168th Street.

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1 department of medicine

2 columbia university

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4 iat1@columbia edu

5 what's new

6 science

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8 ira tabas

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10 reviews

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Ira Tabas, MD, PhD, Columbia University | tabaslab.com Reviews

https://tabaslab.com

Macrophage apoptosis and defective efferocytosis lead to plaque necrosis, a key feature of clinically dangerous atherosclerotic plaques. In the setting of obesity, insulin resistance, and type 2 diabetes, liver-derived metabolic perturbations and direct effects of insulin resistance on lesional macrophages promote plaque progression. To block the generation of systemic, liver-derived atherosclerotic risk factors in the setting of obesity and type 2 diabetes. Ira Tabas, MD, PhD. 630 West 168th Street.

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tabaslab.com

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Ira Tabas, MD, PhD, Columbia University

http://www.tabaslab.com/lab/tabas.htm

Ira Tabas, MD, PhD. Richard J. Stock Professor and Vice-Chair of Research. Professor of Pathology and Cell Biology (in Physiology and Cellular Biophysics). Columbia University Medical Center. Click to view C.V. Requires Adobe Acrobat Reader. Brief Biography of Dr. Tabas:. Journal of Clinical Investigation. Proceedings of the National Academy of Sciences. And other top journals. Dr. Tabas also serves on the Board of Reviewing Editors for the journal. Last updated: May 18, 2016.

2

Ira Tabas, MD, PhD, Columbia University

http://www.tabaslab.com/protocols/index.htm

Tips on Mouse Handling. Assay of Hepatic p-CaMKII in Fasting Mice. Genotyping of Asm-/- Mice. Protocol for Bone Marrow Transplantation. Principles of Mouse Husbandry: George Kuriakose's 2013 PowerPoint Presentation. Assessment of Progression of Atherosclerotic Lesions in Mice. Genotyping of F4/80-Cre Knock-In Mice. Basic Information for Directing a Mouse Atherosclerosis Project. Important Information About the Harlan-Teklad 88137 Western Diet Used in the Tabas Laboratory for Mouse Atherosclerosis Studies.

3

Ira Tabas, MD, PhD, Columbia University

http://www.tabaslab.com/lab/index.htm

Ira A Tabas, MD, PhD. Vice-Chairman of Research, Department of Medicine, Columbia University. Professor of Medicine and Anatomy and Cell Biology (in Physiology and Cellular Biophysics). Columbia University College of Physicians and Surgeons. Faculty Consultant for Lab. Jonathan D. Proto. Xiaobo Wang received his PH.D. degree in 2008 from China Agricultural University in Beijing, China. After three years postdoctoral research working on the smooth muscle gene expression during smooth muscle ce...

4

Ira Tabas, MD, PhD, Columbia University

http://www.tabaslab.com/pubs/index.htm

Search PubMed for articles. Ira Tabas. Death-defying plaque cells. Nature: Vol 536, 4 August, 2016 [ pdf reprint. Lale Ozcan, Devram S. Ghorpade, Ze Zheng, Jane Cristina de Souza, Ke Chen, Marc Bessler, Melissa Bagloo,. Beth Schrope, Richard Pestell, and Ira Tabas. Hepatocyte DACH1 Is Increased in Obesity via Nuclear Exclusion of HDAC4 and Promotes Hepatic Insulin Resistance. 15:1–12, June 7, 2016 [ pdf reprint. Wwwpnas.org/cgi/doi/10.1073/pnas.1524292113,. 1-6 [ pdf reprint. Lale Ozcan, Xiaoming Xu, Shi...

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columbiamedicine.org

Divisions

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Divisions of Internal Medicine. Digestive and Liver Diseases. Fellowship in Hospice and Palliative Medicine. Preventive Medicine and Nutrition. Pulmonary, Allergy/Critical Care Medicine.

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Dennis Goldberg, Ph.D., FAHA. Eric M. Morrel, Ph.D. Phillip M.Friden, Ph.D. John M. (Jock) Holliman, III. Executive Chairman of Capstone Therapeutics. Randolph C. Steer, M.D., Ph.D. - Chief Medical Officer. GM Anantharamaiah, Ph.D. Professor, Department of Medicine, University of Alabama at Birmingham. Michael Davidson, M.D. - University of Chicago, Medical Center. Ira Tabas, M.D., Ph.D. - Department of Medicine, Columbia University. Robert Raffai, Ph.D. Associate Professor of Surgery, UCSF. Oct 14, 2015.

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tabaslab.com

Ira Tabas, MD, PhD, Columbia University

Macrophage apoptosis and defective efferocytosis lead to plaque necrosis, a key feature of clinically dangerous atherosclerotic plaques. In the setting of obesity, insulin resistance, and type 2 diabetes, liver-derived metabolic perturbations and direct effects of insulin resistance on lesional macrophages promote plaque progression. To block the generation of systemic, liver-derived atherosclerotic risk factors in the setting of obesity and type 2 diabetes. Ira Tabas, MD, PhD. 630 West 168th Street.

tabaslab.org

Ira Tabas, MD, PhD, Columbia University

Macrophage apoptosis and defective efferocytosis lead to plaque necrosis, a key feature of clinically dangerous atherosclerotic plaques. In the setting of obesity, insulin resistance, and type 2 diabetes, liver-derived metabolic perturbations and direct effects of insulin resistance on lesional macrophages promote plaque progression. To block the generation of systemic, liver-derived atherosclerotic risk factors in the setting of obesity and type 2 diabetes. Ira Tabas, MD, PhD. 630 West 168th Street.

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